The following is from the Kruse Longevity Center.
Electromagnetic fields can activate voltage-gated calcium channels (VGCCs) via molecular resonance in the plasma membrane of cells. When electromagnetic fields activate these channels, large amounts of intracellular calcium (Ca2+) are produced. Calcium flux controls and links to flows of magnesium via the MRS2 transporter. The amount of calcium produced is subject to the wave physics of the emitted light wave from the point source. Indirectly this shows you how the incident light a cell senses actually control magnesium flows from the cytosol to the matrix as well. If the wave is polarized this also changes the calcium efflux and its ionic resonance. Calcium is the key secondary messenger in cells for these antigens on the surface and they interact and react with RNS species like peroxynitrite (RNS radical).Dr. Jack Kruse via the Kruse Longevity Center@facebook.com
Using electron released from NADH and FADH2 produced in the TCA cycle, a proton gradient is generated across the mitochondrial inner membrane via the electron-transport chain, which results in the generation of mitochondrial membrane potential (ΔΨ). To assess the ΔΨ, researchers have quantified the ratio (red to green) of average mitochondrial fluorescence intensity of 5, 5′, 6, 6′-trichloro-1, 1′, 3, 3′-tetraethylbenzimidazolylcarbocyanine iodide (JC-1) per cells in MRS2 KD HeLa cells. It turns out that the red-to-green ratio of JC-1 fluorescence, indicated the mitochondrial inner membrane potential, was lower than control cells. This result has suggested the mitochondrial Mg2+ uptake through MRS2 is crucial for maintaining the ΔΨ (delta psi). Anything that slows the substrate cycling through the distal TCA intermediates also will slow Mg2+ flow through the cytosol into the matrix via the MRS2 Mg2+ transporter. Deuterium is just one thing capable of doing this. Dehydration of cytosolic water is another. Since Mg2+ mode of action is heavily dependant on being hydrophilic, drinking water can never be an unwise thing when matrix function is disrupted. Anyone who says otherwise is just not as wise as they should be.
Lowering Mg2+ changes coupling rates in the matrix via MRS2 FYI. Calcium efflux or influx controls this process.
This excess calcium within the cells produces a chain of free radical chemical reactions from the mitochondria which change the physiology of the cell at a femtosecond basis. This leads to the production of a VARYING free radical signal that causes organizational chaos in the cell. That chaos is manifested by the disruption of the incident EMF that excites and activates the voltage-gated channels in the cell. A highly variable incident EMF with produce a highly variable ROS/RNS mitochondrial signal and this results in unpredictable biochemistry in the cell. This is associated with the production of chronic oxidative stressor chemicals in the living system that cannot be buffered by the normal quenching chemicals in cells. All of this results from an environment that produces an altered electric and magnetic field around an animal/human.
The chaotic free radical signal is capable of culminating in DNA damage and or the change in the plasma membrane to lead to pathologic symptoms and eventual disease.
The calcium efflux causes excess calcium directly in and around the cell and in its local environment. So with respect to RBCs, it also means that this effect of electric and magnetic fields will also affect the surface of the blood vessels. Peroxiredoxins are the key peripheral circadian controller that removes CpG Island from the fragmented DNA and mtDNA that enter the blood when nnEMF is destroying cellular biology. Tight control of RBC circadian cycles links RBC antigen clearance to the innate immune system via C4. RBC becomes more permeable to toxins in this case and as a result, the RBC ages faster and more antigens pass through the circulatory system. This is really what happens in all mold and biotoxin disease. It is not the mold or toxin that is critical in this case, it is REMOVAL of the nnEMF field that is critical to get right. Most of the clinician out there never get this advice to their patients or the public.
All of these mechanisms of nnEMF field exposure alter melanopsin biology in the blood and arteries to a chaotic release of nitric oxide (NO) within cells and in arteries to cause disease when it occurs chronically and affects mitochondrial function when other frequencies of sunlight are subtracted from this photic dance.
The increase of nitric oxide is a chameleon event in the blood plasma. Endothelial nitric oxide synthetase has a quantum superposition effect on sulfur atoms in the skin, arteries, blood, and gut to protect us from dangerous nitrogenous groups in these antigens.
This means that any pulsed or polarized non-native man-made electromagnetic signal can have a variable non-linear effect on sulfation and nitrosylation pathways in any of these organs. I covered this last night in my Feb 2019 webinar Q & A.
It can result in therapeutic effects or detrimental effects in the blood plasma depending upon the nnEMF stimulus. This will lead to highly variable chaotic mitochondrial energy flux and fidelity signal dynamics. This is very damaging to the matrix and directly affects what biochemistry can or cannot occur. This is one reason why non-thermal electromagnetic fields (PEMF) are increasingly used in medical therapies, but they are being used without any proper understanding of how they truly operate.
Today the sellers and purveyors of these devices think and believe that their RF/microwaves effect is always beneficial therapeutically in a wildly variable world of surrounding nnEMF. THIS IS PURE FALLACY AND MARKETING BULLSHIT.
Moreover, they fail to realize that this eNOS switch in cells is very sensitive to any variable PEMF RF pulse. This is why PEMF devices need to be strictly avoided in a 5G world. Yes, that includes all the Oura rings and PEMF devices pushed by BEMER and Dr. Havas based upon the latest NTP study on RF radiations.
For example, if one is in an environment that fosters chronic nitric oxide release this means there will be a relative lack of sulfation of the skin, arteries, and gut and RBC’s and this would favor the activation of the reactive nitrogen species of chemicals. This is particular devasting to the microbiome because NO and H2S work in unison to control the constitution of the microbiome.
In fact, we now know that nitric oxide can also interact with the superoxide pulse (OO-) created in cytochrome one (NAD+/NADH) form altered mitochondrial function to create peroxynitrite (ONOO-) to do further damage. This is why people with gut and microbiome conditions relapse so often in toxic nnEMF environments loaded with blue light. Most of the doctors are not sophisticated enough yet to understand that things like SIBO and adrenal fatigue are adaptative and not pathologic symptoms tied to altered and highly variable EMF fields that their patients live in.
It has been found that when peroxynitrite breaks down, it creates reactive free radicals and oxidative stress within cells and this likely leads to many of the symptoms of CV and neurodegeneration on longer timescales. In this way, both atherosclerosis, CV, and neurodegeneration can be thought severe chronic adaptive mechanisms employed by cells who have developed an innate immune allergy to nnEMF. https://www.youtube.com/watch?v=bsaB7ewFsN0
Here at the Kruse Longevity Center we aim to teach you to renovate yourself back to whole using quantum biologic cutting edge materials utilizing light, water, and magnetism all tied to natures fundamental rules to build wisdom inside of you that you came into this world with.KLC